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P.I


Tobias Meyer

Ca2+, lipid second messenger and small GTPase signaling pathways; Control of cell polarity, chemotaxis, and collective migration as well as cell proliferation and differentiation decisions

email: tobias1 at stanford.edu


Postdocs


Lin Gan

My research interests are understanding mTOR signaling and nutrient sensing from a global perspective as well as elucidating the mechanism of nutrient sensing at the molecular level.

email: lgan at stanford.edu


Damien Garbett

In order to migrate as a cohesive group, cells must coordinate their polarization signals, migratory direction and speed, maintenance of cell-cell junctions, and cytoskeletal activity all at both the single cell and group levels. I am interested in how cells within a group are able to achieve this remarkable level of coordination.

email: dgarbett at stanford.edu


Arnold Hayer

Collective cell migration is required during development, for morphogenesis, and for repair following injury. Directional signals guiding collective cell movement are thought to be transmitted mechanically between cells via cell-cell junctions. Using monolayers of primary human endothelial cells (HUVEC) as a model system, I study how forces applied to the junction locally by one cell are sensed by its neighbor and converted into biochemical signals.

email: arnoldh at stanford.edu


Marielle Köberlin

The composition of the cellular lipid landscape is essential for the regulation of biological processes. Previously, the functional annotation of membrane lipids in macrophages led to the identification of lipid species differentially regulating receptor signaling. Cell migration and cell division are both fundamental processes that are precisely regulated by conserved signaling pathways combined with the availability of metabolites. My research is centered around the identification of metabolic pathways and specific metabolites regulating cell cycle progression and migration.

email: mkoeberlin at stanford.edu


Lindsey Pack

I am interested in understanding how distinct chromatin modifications regulate the entry, exit, and maintenance of quiescence through control of both chromatin accessibility and site-specific transcriptional control of critical cell cycle genes.

email: lpack at stanford.edu


Hee Won Yang

I am interested in understanding the signaling network in systems level. How signaling proteins induce signaling cascades, cross-regulate, and are correlated with each other.

email: heewony at stanford.edu


Graduate students


Anjali Bisaria

I study how asymmetry cytoskeleton organization promotes and maintains morphological and signaling polarity during cell migration. Specifically, I am interested in how the actin cortex and associated ERM proteins alter membrane tension.

email: abisaria at stanford.edu


Mingyu Chung

The rate of cell division is largely dictated by the amount of time individual cells spend in a non-dividing state before entering a cycle of cell division. My research focuses on dissecting the signaling pathways regulating cell-cycle entry.

email: mingyuc at stanford.edu


Leighton Daigh

My research focuses on how stochastic levels of endogenous cellular stress impact cell cycle dynamics and the proliferation decision.

email: ldaigh at stanford.edu


Yilin Fan

I study the transcriptional regulation of cellular quiescence and cell-cycle entry.

email: yilinfan at stanford.edu


Katie Ferrick

Research interests TBD.

email: kferrick at stanford.edu


Bo Gu

Joint with the Wysocka lab

email: bgu1224 at stanford.edu


Chad Liu

I am interested in understanding the mechanism underlying hysteresis in cell cycle entry.

email: chadliu at stanford.edu


Nalin Ratnayeke

I study regulatory dynamics at the G1/S transition, particularly how cells control DNA replication machinery in order to faithfully duplicate their genome.

email: nalinratnayeke at stanford.edu


Admin


Jennie Rivera Visitacion

Administrative Associate

email: jenniev at stanford.edu


Jaime Larios

Lab technician